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310 Similar effects are observed after transgenic deletion of Ccl2 specifically from astrocytes, which showed little effects early in EAE but demonstrated that astrocyte Ccl2 sustains disease symptoms and inflammation during chronic EAE, making it a therapeutic target

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Chronic type I IFN Is sufficient to promote immunosuppression through accumulation of myeloid-derived suppressor cells

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10.1038/s41416-022-01868-6 63 HeynH.EstellerM

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[DOI] [PMC free article] [PubMed] [Google Scholar] 151.Denko NC

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Mitochondrial pharmacology

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