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MiR-34 was found to reduce neuronal viability through mitochondrial dysfunction and the production of ROS in vitro (Minones-Moyano et al., 2011
Zhonghua Yu Fang Yi Xue Za Zhi
doi: 10.1089/ars.2011.3968
Furthermore, at the high expression levels used, GHSR1a homodimerization may be artificially enhanced
