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Evidently, intracellular cyst(e)ine contribute to ferroptosis suppression via at least two additional pathways in different contexts: in a mouse lung adenocarcinoma model, NFS1 cysteine desulfurase utilizes cysteine to synthesize ironsulfur clusters, which are necessary for preventing activation of the iron-starvation response, subsequent iron influx and ferroptosis 14 (Fig

Injection sites can be local (near injury) or systemic (abdominal or thigh)

Some studies have suggested utilizing vitamin E in combination with other therapeutic agents, particularly with vitamin C, due to their synergistic or additive therapeutic effects

321 Consequently, effectively eradicating CSCs requires strategies that address the tumor hierarchy, resistance mechanisms, metabolic vulnerabilities, and targeted immunotherapies

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