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Description
In addition, we report that the same pharmacological treatment rescues PPI deficits in Tbx1 mutants, suggesting that they are related to the underlying metabolic changes caused by the mutation
Angiogenesis and endothelial modulation: BPC157 has been reported in multiple preclinical studies to upregulate angiogenic pathways, including increased expression and activity of vascular endothelial growth factor (VEGF) and related downstream signaling (e.g., VEGFR2 activation, modulation of angiogenic transcriptional programs)
In vitro, BPC-157 promotes fibroblast migration, angiogenesis, and nitric oxide (NO) pathway modulation, supporting endothelial cell survival and wound repair
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