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[DOI] [PMC free article] [PubMed] [Google Scholar] 69.Pereira C.P.M., Souza A.C.R., Vasconcelos A.R., Prado P.S., Name J.J

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Incubation of -synuclein monomers with cytochrome c/H 2 O 2 led to -synuclein aggregation in vitro by catalyzing the crosslinking of -synuclein tyrosine residues through 3,3-dityrosine bond formations [91, 92]

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However since the TrxR1 mutation is liver-specific, the question arises: could other cells in these mice, cells that do have TrxR1, supply reducing power to the TrxR1/Gsr-null livers