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However, under pathological conditions, excessively activated NSPs become major drivers of tissue destruction, contributing to the development and progression of various inflammatory lung diseases ( The three primary NSPs exhibit both shared and distinct functional characteristics

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doi: 10.1016/j.agee.2012.12.006 43 WeiC.MaL.ChengY.GuanY.GuanJ

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doi: 10.1016/j.pnpbp.2008.10.009

C3 protein is cleaved and activated by C3-convertase to C3a and C3b in the complement system 4 ) and apoptosis 5