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Why did the lower dose work better than the higher dose

Histopathology of nonalcoholic fatty liver disease/nonalcoholic steatohepatitis

Researchers have found that SFN inhibits histone deacetylase (HDAC) activity and enhances acetyltransferase expression, thus further causing DNA damage

-glutamyl kinase (GK) encoded by PRO1 in eukaryotes or proB in prokaryotes, the first enzyme in proline biosynthetic pathway, catalyzes the formation of intermediate -glutamyl phosphate, which if accumulated could partially react with cysteine to generate -GC, and then Gsh2 adds glycine to -GC to form GSH

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