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In genetically obese mouse models, there is an increase in intestinal permeability and plasma endotoxins and proinflammatory cytokines, such as interleukin one beta (IL-1), interleukin-6 (IL-6), interferon-gamma (INF), and tumor necrosis factor (TNF-), compared to wild-type mice

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This process occurs at normal LDL concentrations but is enhanced when LDL concentrations are increased and by modification (e.g

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