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This section summarizes the current research status of reported ferroptosis-related pharmacological agents, analyzing their mechanisms of action and therapeutic effects in AD models, aiming to provide theoretical foundations and structural references for future anti-AD drug design based on ferroptosis mechanisms ( Table 1 )

It is related to oxidative stress damage in cerebrovascular diseases

For infection experiments, pFK i389 JcR2A 38 , pFK i389 JcR2A GDD, pFK i389 JcR2A E1E2 were used

For cagrilintide, readers may see trial references, research-peptide marketing, or forum posts

Acta 491, 8590

This explains why standard protocols use moderate doses rather than attempting to maximize peptide exposure