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Although APX activity has been reported in a number of non-plants, it appears that a fundamental difference between mammals and plants is an additional high capacity H 2 O 2 ascorbate-based scavenging system [2], [22] in the latter to complement catalase as well as the more widely conserved peroxiredoxin and glutathione peroxidase-like systems [23], [24]

Further longitudinal clinical studies and targeted interventional trials are necessary to determine whether modulating microglial activity or ferroptosis can meaningfully alter the disease course in AD patients

Today, microfluidic devices make it possible to assess RBCs deformability, aggregation, and hematocrit simultaneously 42

Cell 93 (2), 189201

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