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Blood (2014) 123(17):266372

Results: The pathogenesis of DMED encompasses vascular dysfunction, endothelial cell damage, cavernous smooth muscle defects, neurological dysfunction, endocrine/metabolic factors, leukomalacia fibrosis, and psychosocial factors, elucidating complex interplay among the mechanisms underlying DMED

Gao X, Chen Q, Yao H, Tan J, Liu Z, Zhou Y, et al

Glutamate transporter-associated anion channels adjust intracellular chloride concentrations during glial maturation

Solubilization of tomato fruit pectins by ascorbate: a possible non-enzymatic mechanism of fruit softening

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