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from George Mason University School of Law.

Our findings reveal that TFRC and ACSL1 are functionally distinct yet targetable nodes that govern ferroptosis vulnerability in CLL patients and may guide novel therapeutic strategies for high-risk patients.

This review synthesizes clinical data, genetic studies, and therapeutic interventions across experimental models to establish ferroptosiss multifaceted involvement in diabetes progression

Current research priorities include large-scale studies to better understand the tissue-specific effects of NNMT inhibition and the temporal dynamics of metabolic reprogramming following treatment

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