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Postnatally, POU3F4 knockdown also leads to apoptosis of SGNs, but does not appear to result in changes in the proportion of SGN isoforms [70]

demonstrated a significant reduction in neuronal damage in the CA1 area of the hippocampus by administering Lin as a TAC modulator after cerebral ischemia (Xu et al., 2018)

Monyer H, Burnashev N, Laurie DJ, Sakmann B, Seeburg PH

2023YJXK01), Fund of Yangpu Hospital Affiliated to Tongji University (No

It is commonly believed that enzyme active-site loops provide the necessary plasticity so that the active site can adopt different conformations during the substrate entrance, catalysis, and product release

Table 1 provides a gist of the mechanisms by which genetic polymorphisms influence prooxidant genes
