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mTORC1 and mTORC2 converge on the arp2/3 complex to promote kras(g12d)-induced acinar-to-ductal metaplasia and early pancreatic carcinogenesis

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This product of the hexosamine pathway competes with phosphorylation at post-translation modification sites on transcription factors and disrupts the regulation of inflammatory responses that generally occurs through transcription growth factor- (Yumnamcha et al., 2020)

In general, butyrates inhibitory effect was seen in apolipoprotein B-48 output, TG export and chylomicron and VLDL secretion (79)