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But luteolin also supports healthy aromatase activity, and those two jobs are directly connected
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NOX 2 -dependent signaling promotes several deleterious processes in cardiac pathology, including cardiomyocyte hypertrophy, contractile dysfunction, interstitial fibrosis, and cell death
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Overexpression of HDAC2 in neurons induces a decrease in dendritic spine density, synaptic number, synaptic plasticity, and memory formation in mice
