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The same was observed in clinical CD patients and in trinitrobenzene sulfonic acid (TNBS)-induced experimental colitis: increased expression of ACSL4/MDA/PTGS2/iron and inhibition of GPX4, which was ameliorated by treatment with Ferr-1 (63)
( A and B ) GPX4 expression level was evaluated by flow cytometry in activated WT and adora2a fl/fl Lck cre cKO CD8 + T cells
The absence of PDIA6 was recently demonstrated to enhance ER stress and ferroptosis in proximal tubule epithelial cells (Kim et al
impaired immune responses that increase susceptibility to infections
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