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Collectively, the data point toward potential modulation of focal adhesion kinase-related pathways and paxillin-associated signaling involved in F-actin assembly

Now, as for the reversibility of nerve damage: In terms of neurological recovery from B12 deficiency, studies conducted on rats so far indicate that methyl B12 may promote the regeneration of nerves: Schwann cells (SCs) are constituents of the peripheral nervous system

doi: 10.1038/s42255-022-00710-w 49 Martnez-ReyesIChandelNS

Therefore, the side effects and safety of this peptide have not yet been fully explored

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