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The initial mechanical disruption of white matter tracts is rapidly exacerbated by multiple secondary sequelae that include ischemia, inward calcium flux, infiltration with inflammatory cells and liberation of high levels of ROS which evolve over time to produce a chronic state of inflammation

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The Association Between Serum Glutathione Peroxidase-3 Concentration and Risk of Acute Kidney Injury After Cardiac Surgery: A Nested Case-Control Study

Moreover, pro-inflammatory cellular-stress pathways can be homeostatic when activated transiently or moderately and when they remain below the threshold for overt inflammation

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