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Mitochondrial dysfunction was also observed, with enhanced mitophagy and reduced fusion, resulting in low ATP production, complex IV, and ATP synthase levels [106]

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1999a;121:11691177

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Bone 2008;43:869-879

Experimental investigations have examined interactions between glutathione-dependent redox systems, Nrf2-associated signaling mechanisms, and the regulation of enzymes involved in glutathione biosynthesis and utilization