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Notably, this compound exhibits protective effects on normal cells (59) and is being investigated as a potential therapeutic agent for neurodegenerative and aging-related conditions (60, 61)

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Deletion of egtA and yap1 and complementation of the egtA and egtA yap1 strains For generating egtA and yap1 deletions in AfS77, and the associated double mutants, the bipartite marker technique was used 61

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