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Zinc accumulation induces ISR to initiate -cell fate in cells via the zinc-ATF4-ARX axis Gene set enrichment analysis (GSEA) of differentially expressed genes (DEGs) in SC- cells from the excessive-zinc group versus the control group revealed that pathways associated with -cell function, including regulation of gene expression in cells, regulation of -cell development, and regulation of insulin secretion, were significantly downregulated in the excessive-zinc group

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