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Furthermore, the downregulation of NF-B expression in the main cochlear structures has been proposed as underlying mechanism of the protective effect of CCM against cisplatin-induced ototoxicity (Paciello et al

Overall, G6PDX mice provide a clinically relevant model of G6PD deficiency that reflects the extent of limited G6PD activity commonly found in human patients with G6PD deficiency (6, 12)

This is achieved through the activation of pro-survival signaling pathways, including the Phosphoinositide 3-kinase (PI3K)/Akt pathway

Similarly, GRO, a SASP component induced by oncogenic RAS, encourages the senescence of normal human ovarian fibroblasts [216]