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Although the mechanism of pathogenesis is unclear, it is thought that the clinical phenotypes of SSMD patients involve complete loss of GPX4 enzymatic function

Its not at all clear

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In SHRs, GA significantly reduced the expression of four CaMKII isoforms: , , , , and the expressions of caspase-3, Bax, p53, and p300 proteins, thereby reducing angiotensin II-induced angiotensin II-induced apoptosis (Jin et al., 2018)

This resonates strongly with our recent European Heart Journal study (Baka et al., EHJ 2025), where metabolic modulation with SGLT2 inhibition improved energetic efficiency, contractile balance, and hypertrophy