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Importantly, circulating LCAC levels were found to normalize to non-failing levels following left ventricular assist device implantation of aortic valve replacement (Shah et al., 2010, 2012)

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Firstly, as will be discussed below, LCACs induce Ca 2+ overload within cardiomyocytes via release of sarcoplasmic reticulum (SR) Ca 2+ and inhibition of mitochondrial Ca 2+ uptake (Yamada et al., 2000)

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These findings provide the first evidence that the previously observed increases in NO with GPLC may be associated with performance improvements in trained individuals