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The evidence is confined to a limited set of animal studies, with no human genetic or interventional data to establish a causal role in MASLD

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D-galactose induces damage to hippocampal neurons by increasing ROS production and advanced glycation end product accumulation, leading to oxidative stress, mitochondrial dysfunction, and neuroinflammation

Alternatively, SIRT1 enhances HSF-1 expression and prolongs its activation by binding to the promoters of HS genes, leading to the HSPs expression [94,158]