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[DOI] [PMC free article] [PubMed] [Google Scholar] 14.Martic I., Wedel S., Jansen-Drr P., Cavinato M

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comparing the Kras -mutant LUAD mouse models with p53 knockout, WT p53 or hyperactive p53 alleles with enhanced tumour suppressor activity has revealed the role of p53 loss in the initiation and progression of LUAD

1F, G), contrary to previously reported findings, where colchicine improved the relative protein expression of KU70/KU80, which was decreased in endothelial cells treated with H 2 O 2 and ethanol [26, 27]

Therefore, the upregulation of these genes by p53 possibly serves to sensitise cells to apoptosis by cytotoxic T cells and natural killer (NK) cells, which can express the ligands for death receptors