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cigarette smoke induces cytoplasmic p53 accumulation Tobacco condensate-induced senescence in endothelial cells was ameliorated by colchicine treatment via suppression of NF-κB and MAPKs P38 and ERK pathways activation | Cell Communication and Signaling Chronic Cigarette Smoke-Induced Epigenomic Changes

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Notably, miR-122 can exert a protective function in APAP-induced hepatotoxicity by modulating CYP1A2 and CYP2E1 (Chowdhary et al., 2017)

cigarette smoke induces cytoplasmic p53 accumulation Tobacco condensate-induced senescence in endothelial cells was ameliorated by colchicine treatment via suppression of NF-B and MAPKs P38 and ERK pathways activation | Cell Communication and Signaling Chronic Cigarette Smoke-Induced Epigenomic Changes

D.MitchelE

cigarette smoke induces cytoplasmic p53 accumulation Tobacco condensate-induced senescence in endothelial cells was ameliorated by colchicine treatment via suppression of NF-B and MAPKs P38 and ERK pathways activation | Cell Communication and Signaling Chronic Cigarette Smoke-Induced Epigenomic Changes

Photoprotection of human skin beyond ultraviolet radiation

cigarette smoke induces cytoplasmic p53 accumulation Tobacco condensate-induced senescence in endothelial cells was ameliorated by colchicine treatment via suppression of NF-B and MAPKs P38 and ERK pathways activation | Cell Communication and Signaling Chronic Cigarette Smoke-Induced Epigenomic Changes

Research Square

cigarette smoke induces cytoplasmic p53 accumulation Tobacco condensate-induced senescence in endothelial cells was ameliorated by colchicine treatment via suppression of NF-B and MAPKs P38 and ERK pathways activation | Cell Communication and Signaling Chronic Cigarette Smoke-Induced Epigenomic Changes

Upon UVR-induced DNA damage, different pathways lead to the dissociation of the p53/mouse double minute 2 (MDM2) inhibitory complex [28] and two major kinases, ataxia telangiectasia mutated (ATM) and ataxia telangiectasia and Rad3-related (ATR), mediate p53 phosphorylation, preventing its degradation [29]

cigarette smoke induces cytoplasmic p53 accumulation Tobacco condensate-induced senescence in endothelial cells was ameliorated by colchicine treatment via suppression of NF-B and MAPKs P38 and ERK pathways activation | Cell Communication and Signaling Chronic Cigarette Smoke-Induced Epigenomic Changes

It further addresses the interaction between p53 and vitamin D receptor (VDR), which may aid in suppressing skin carcinogenesis

cigarette smoke induces cytoplasmic p53 accumulation Tobacco condensate-induced senescence in endothelial cells was ameliorated by colchicine treatment via suppression of NF-B and MAPKs P38 and ERK pathways activation | Cell Communication and Signaling Chronic Cigarette Smoke-Induced Epigenomic Changes

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