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Taken together, understanding the contributions of immune and metabolic parameters to tumour development is crucial for improving the predictive value and therapeutic strategies for p53 mutant lung cancers
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Recently, a p53 -dependent gene ring finger protein 144b (Rnf144b ) has been found to be involved in the repair of DNA DSBs and is shown to have a pivotal tumour suppressive function, particularly in lung adenocarcinoma [57]
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