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& Vousden, K

In recent years, evidence has suggested that epigenetic modification mechanisms can possibly explain the link between CS exposure and inflammation

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Cell Dev

CS increased the expression of pro-inflammatory mediators IL - 8 , TNF - and matrix metalloproteinase 9 through HDAC1 depression in rat lungs and in macrophages, while knocking down HDAC1 increases the expression of these genes [37]