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Additionally, high plasma homocysteine levels are a risk for deep venous thrombosis in the general population (den Heijer et al, 1996)

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Therefore, the discovery of marker molecules of autophagy-associated death and related metabolic mechanisms, the exploration of clinically applicable target drugs, sensitive indicators of autophagic flux and autophagy outcomes, and the application of these markers in combination with autophagy transitioning in the clinic may transform disease treatment

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