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Interestingly, blocking these receptors in control rats treated with saline did not affect neuronal activity

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Through this integrated in vivo and organoid-guided approach, our study aims to unravel how chronic smoke exposure disrupts lung stem cell function and regenerative capacity, providing deeper mechanistic insights into COPD progression and highlighting potential cellular and molecular targets for regenerative therapy

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This study was included in a meta-analysis of 15 CHANTIX efficacy trials in various clinical populations that showed an increased hazard ratio for Major Adverse Cardiovascular Events (MACE) of 1.95