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UVR also produces excessive amounts of reactive oxygen species (ROS) that can overwhelm the intracellular antioxidant molecules and activate signaling pathways related to inflammation and carcinogenesis [16]

Reviewing the future of the P53 field

A common theme unites these two lineups

Concentration that reduced the expression of miR-34a-5p without toxicity were selected

comparing the Kras -mutant LUAD mouse models with p53 knockout, WT p53 or hyperactive p53 alleles with enhanced tumour suppressor activity has revealed the role of p53 loss in the initiation and progression of LUAD