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Based on the observation that Keap1 functions as a homodimer, the heterozygous missense mutation generates three types of Keap1 dimers, i.e., wild-type homodimer, wild-type-mutant heterodimer, and mutant homodimer at a ratio of 1:2:1

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TABLE 1 Clinical summary of antioxidant intervention in ASD: Population, dose/route, duration, clinical domains, redox biomarkers, safety, and level of evidence are reported for each study