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Understanding the mechanisms underlying METH-induced neurotoxicity is essential for developing effective treatment strategies to mitigate its devastating effects

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e ., bromocriptine, amantadine) and gastric pentadecapeptide BPC 157 could antagonize these lesions, but other antiulcer agents (atropine, pirenzepine, misoprostol, pantoprazole, lansoprazole, cimetidine and ranitidine) were not as effective[10]