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In this regard, it has been shown that tamoxifen, a type of selective estrogen receptor modulator (SERM) that acts as a competitive antagonist or as a partial agonist depending on the cell type, favors the repair of demyelinated lesions in the CNS by increasing the mRNA levels of myelin basic protein (MBP) and 2′, 3′-Cyclic nucleotide 3′-phosphodiesterase (CNP), two genes highly expressed during oligodendroglial differentiation (Table 1)