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Non-alcoholic fatty liver disease (NAFLD) / non-alcoholic steatohepatitis (NASH): Because of its role in lipid metabolism and energy storage, GIP modulation is explored for liver fat accumulation and metabolic liver disease

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In other words: Receptor overstimulation from rapid dose increases is the primary driver of gastrointestinal adverse events, not the compound itself when used therapeutic doses

Dopamine D2-like receptors are expressed in pancreatic beta cells and mediate inhibition of insulin secretion