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Type 1 diabetes and thyroid autoimmunity in children

Deactivation Mechanism of Glucagon-like Peptide-1/2 by Dipeptidyl Peptidase 4 Intestinal L-cells and K-cells release the physiologically active forms of GLP-1 and GIP, known as active GLP-1 amide and GIP, in response to oral glucose via increased cAMP (69)

Broadening use beyond diabetes in tackling comorbidities Perhaps most transformative is that GLP-1 therapies are no longer confined to diabetes alone

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Allosteric compounds such as BETP or compound 2 likewise can augment insulin secretion, albeit that this is complicated by endogenous circulating peptides

Ethical approval The study was reviewed and approved by the Institutional Review Board (IRB) of Hayatabad Medical Complex, Peshawar