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Finally, we outline the implications of adopting semaglutide as a disease-modifying agent in MASH with F2-F3 fibrosis, note the study limitations and remaining questions, and argue that combinatorial approaches targeting complementary metabolic and hepatocentric pathways may define the next phase of MASLD management

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used a proteomic approach to identify a substantial number of potential OGTbinding proteins, revealing an enrichment of proteins involved in the proteolytic degradation pathway