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The lateral parabrachial nucleus may be a critical site of action as pharmacological activation of GLP-Rs in the lateral parabrachial nucleus (LPBN) inhibits feeding whereas antagonism of LPBN GLP-1Rs induces hyperphagia (Alhadeff et al., 2014)

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The PASADENA study and its subsequent TTE analysis have opened up a promising avenue for evaluating disease-modifying therapies in PD

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