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Bile acids promote glucagon-like peptide-1 secretion through TGR5 in a murine enteroendocrine cell line STC-1

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In this context, it is crucial to identify affordable and reliable biomarkers of neurodegeneration and neuroplasticity that can assist in clarifying the underlying mechanisms associated with STN-DBS use, discerning whether they predominantly arise from natural disease progression, the effects of DBS surgery itself, or the direct influence of STN stimulation