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This extensive review demonstrates how GLP-1 RAs have transcended traditional therapeutic boundaries by modulating core cellular mechanisms, including mitochondrial enhancement, anti-inflammatory actions, autophagy-mediated quality control, and neuroprotective pathways that underlie multiple disease states

64,93 Structurally, lixisenatide is based on the exenatide structure but lacks proline at position 38, and six lysines are linked after serine at position 39