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Taken together, with the overlap between OA risk factors and the metabolic syndrome, the demonstrated anti-inflammatory activities of GLP-1 in several tissues [10] and the expression of GLP-1R in joint tissues, GLP-1 analogues may be good therapeutic candidates for treating OA

When administered s.c., liraglutide fails to inhibit eating in mice possessing a CNS-specific deletion of GLP-1R [688]

Liraglutide also has an anticonvulsant effect [73]

Dysregulation of incretin function has been implicated in the pathogenesis of metabolic disorders such as obesity and type 2 diabetes mellitus (T2DM) [1]

Any teen prescribed GLP-1 therapy should receive monthly clinical assessments, lab monitoring (pancreatic enzymes, thyroid function), and family-based behavioral support alongside pharmacotherapy