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These studies demonstrate that there is none or very little GLP-1R expression in the renal vasculature and GLP-1-induced renal artery vasodilation leading to increases in renal blood flow and glomerular filtration rate is diminished in spontaneously hypertensive rats [56, 57]

In conclusion, MNs/GLP-1RAs@NPs-WYRGRL can serve as an efficient drug delivery system to achieve sustained GLP-1RAs release at the disease site and prolong the drug effect

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