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It is currently being evaluated in clinical trials

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showed that in mouse colon smooth muscle cells stimulated with lipopolysaccharide (LPS), exendin-4 significantly reduced the expression of inflammatory cytokines and chemokines, including TNF-, IL-1 (a central mediator of innate immunity and inflammation), TCA-3 (chemotactic factor for neutrophils and macrophages), SDF-1 (chemotactic factor for lymphocytes and monocytes), and M-CSF (controls the survival, proliferation, and differentiation of macrophages), as analysed via antibody array membrane, ELISA, and real-time PCR [24]

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More recently, metabolic cage studies have confirmed that the increased oxygen consumption and energy expenditure effect of OXM is mediated through glucagon receptor signalling (39, 40)