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However, the wide distribution of GLP-1 receptor (GLP-1R) in numerous cells prompted the development of the studies investigating possible effects of GLP1 on different organs [33]

While gastrointestinal issues are common side effects, the biggest setbacks happen when the patient stops taking a GLP-1

Impairment of synaptic plasticity and memory formation in GLP-1 receptor KO mice: Interaction between type 2 diabetes and alzheimers disease

This initiates a cascade that leads to the activation of protein kinase A (PKA), closing of potassium channels, depolarization, and release of calcium from the endoplasmic reticulum.3 An increase of intracellular calcium levels within the cell initiates insulin expression in the nucleus, stimulates energy (ATP) production in the mitochondria, and triggers the release of insulin into the blood stream in a glucose dependent manner (image adapted from Adipogen Life Sciences).4,5 Several commercially and prescribed agents are now available for the management of obesity and type 2 diabetes

Some probiotic strains increase the bodys natural production of GLP-1

Yet these medications are not without drawbacks