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This GLP-1 stimulatory peptide was shown to have sequence homology to Hld from Staphylococcus aureus (denoted Hld Sa ), a phenol soluble modulin that forms a multimeric complex in cell membranes 16

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15 demonstrating powerful inhibition of food intake after ICV administration of GLP-1, an effect that could be blocked by the specific receptor antagonists exendin 939, which also enhanced spontaneous as well as neuropeptide Y-stimulated food intake

The resulting decrease in distal sodium chloride delivery promotes renal hemodynamic dysfunction by impairing the tubulo-glomerular feedback (TGF)

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