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These assets aim to move beyond the current weekly injection paradigm, signalling a future where extended dosing intervals could become central to maintenance strategies [16].

Various GLP-1RAs have been demonstrated to have significant benefits in in vitro and in vivo models of neurodegenerative diseases through modulating a variety of pathogenic mechanisms, including neuroinflammation, autophagy, mitochondrial dysfunction, and the abnormal phosphorylation of pathognomonic proteins

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these signals are hypothesis-generating and should not be interpreted as confirmed causal risks

Results Twenty-eight RCTs were ultimately included for analysis, involving a total of 77,485 participants

Stopping GLP-1 therapy triggers predictable metabolic shifts within weeks