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In addition, this review discusses the multilevel pathophysiological mechanisms involved in acute pancreatitis, including aberrant enzymatic activation, calcium overload, impaired autophagy, inflammatory responses, and various forms of pancreatic acinar cell death

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The potent GLP-1 receptor antagonist, exendin-[939] (Ex9), blunted improvements in glucose tolerance in linagliptin-treated control mice and in GcgRAPDX1Cre mice

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