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The primary anti-inflammatory mechanisms include the inhibition of JAK-STAT and NF-B signaling pathways in keratinocytes, enhanced keratinocyte barrier function through increased expression of tight junction proteins, reduced Th17 polarization that drives psoriatic pathology, and reduced oxidative stress through enhanced expression of antioxidant enzymes [131,139,140]

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Implications for Endometriosis Semaglutides metabolic and anti-inflammatory effects suggest potential benefits for endometriosis, although further research is necessary: Reducing Pain: By lowering inflammation and influencing brain pain pathways, semaglutide may reduce chronic pain from endometriosis

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