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Interestingly, these incretin-sensitising effects of metformin appear to be mediated by a peroxisome proliferator-activated receptor -dependent pathway, as opposed to the more commonly ascribed pathway of metformin action involving AMP-activated protein kinase

Further country-level analyses were conducted to evaluate the overall adoption of testing models in that region

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GIPR agonists have not been utilised in the clinic, hence less is known about the potential effects of manipulating GIPR signalling in the context of cancer

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Deficiency of ICC survival factors (insulin and IGF-1 promote the production of smooth muscle cell-produced stem cell factor, an important ICC survival factor) is detrimental to ICC [60]